Gemella morbillorum

   RANK: Species

TAXONOMY: Firmicutes -> Bacilli -> Bacillales -> Bacillales incertae sedis -> Bacillales Family XI. Incertae Sedis -> Gemella -> Gemella morbillorum

OVERVIEW:

This species was formerly known as Streptococcus morbillorum but was moved to Gemella on the basis of phylogenetic analysis based on 16S rRNA sequence comparisons.Facultative anaerobe producing small (Found in dental plaque, on the tongue and has been isolated from periodontal pockets in periodontitis and exudate from pericoronitis associated with erupting third molarsAlthough found as part of the normal microbiota of the mouth and gastrointestinal orogenital tracts, Gemella morbillorum is capable of causing a wide range of infections. Most of these can be attributed to spread via the bloodstream and thus infective endocarditis is the commonest. Other infections include meningitis, brain, liver and lung abscess, osteomyelitis and septic thrombophlebitis.Identified as a constituent of the oral microbiome by Human Oral Microbiome Database. Identified as constituent of vaginal microbiome. [PMID:23282177]

This species has been identified as a resident in the human gastrointestinal tract based on the phylogenetic framework of its small subunit ribosomal RNA gene sequences.[PMC 4262072]

COGEM
COGEM released a comprehensive database of pathogenicity assessment of around 2575 bacterial species in 2011. The database ranks the pathogenicity of species on a scale of 1 to 4. Gemella morbillorum ranks on this scale:


TAGS
Keystone Core species Type species Pathogen Dysbiosis associated Flora/ commensal Gut associated Probiotic
Leanness Obesity Skin microbiome Fecal distribution Oral microbiome Vaginal microbiome Butyrate producer Catalase producer
Histamine producer Food fermenter Amylolytic Propionate producer Nitrifying Biofilm producer
INTERACTIONS
KEGG PATHWAYS

CLUSTERS WITH
METABOLOMICS       
NUTRIENTS/ SUBSTRATES
  • D-Glucose [parent]

  • ENDPRODUCTS
  • CO2 [parent]
  • Acetic acid [parent]
  • Lactic acid [parent]

  • INHIBITED BY

    ENHANCED BY

    BIOTRANSFORMS

    BIOTRANFORM
    ANTIBIOTIC RESISTANCE   BIOFILM FORMERS   COGEM PATHOGENICITY   

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