Citrobacter
RANK: Genus
TAXONOMY: cellular organisms -> Bacteria -> Proteobacteria -> Gammaproteobacteria -> Enterobacteriales -> Enterobacteriaceae -> Citrobacter
OVERVIEW:
Straight rods, ~1.0 µm × 2.0–6.0 µm. Occur singly and in pairs. Conform to the general definition of the family Enterobacteriaceae. Usually not encapsulated. Gram negative. Usually motile by peritrichous flagella. Facultatively anaerobic, having both a respiratory and a fermentative type of metabolism. Grow readily on ordinary media. Colonies on nutrient agar are generally 2–4 mm in diameter, smooth, low convex, moist, translucent or opaque, and gray with a shiny surface and entire edge. Mucoid or rough forms may occur occasionally. Oxidase negative. Catalase positive. Chemoorganotrophic. Citrate can be utilized as a sole carbon source by most strains. Lysine is not decarboxylated. Alginate and pectate are not decomposed. d-glucose is fermented with the production of acid and gas. The methyl red test is positive; the Voges–Proskauer test is negative. Occur in the feces of humans and some animals; probably normal intestinal inhabitants. Sometimes pathogenic and often isolated from clinical specimens as opportunistic pathogens. Can also be found in soil, water, sewage, and food.The mol% G + C of the DNA is: 50–52 (Tm).Type species: Citrobacter freundii
This genus contains microbial species that can reside in the human gastrointestinal tract. [PMC 4262072]
Microbial Abundance Data: Citrobacter (Percent of total population with standard deviation [PMID: 22698087]) |
Group 1 | Group 2 | Group 3 | Group 4 |
Group 1 Avg | Buccal Mucosa | Keratinized Gingiva | Hard Palate |
Group 2 Avg | Throat | Throat | Tonsils | Saliva |
Group 3 Avg | Supragingival Plaque | Subgingival Plaque |
Stool |
0.002 % (0.016) |
0.001 % (0.004) |
0.001 % (0.006) |
0.006 % (0.038) |
0.002 % (0.011) |
0.004 % (0.029) |
0.001 % (0.008) |
0.000 % (0.001) |
0.001 % (0.007) |
0.001 % (0.011) |
0.000 % (0.002) |
0.002 % (0.020) |
0.000 % (0.006) |
TAGS
DESCENDANTS
|
INTERACTIONS
|
KEGG PATHWAYS
CLUSTERS WITH
| METABOLOMICS   
  
| ANTIBIOTIC RESISTANCE    | BIOFILM FORMERS    | COGEM PATHOGENICITY    |
|